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The vitreous is a clear liquid that fills our eyes and gives them shape. When we are young, the vitreous has a thick, gelatinous consistency and is firmly attached to the retina. As we age, the vitreous thins and separates from the retina. Although this usually results in nothing more than a few harmless floaters, tension from the detached vitreous can sometimes tear the retina.
If liquid seeps through the tear and collects behind the retina or between its nerve layers, the retinal tear can become a retinal detachment. Retinal detachment can cause significant, permanent vision loss and requires immediate medical treatment.
There are three kinds of retinal detachment. The most common form, described above, occurs when fluid leaks into the retina; people who are nearsighted or who have had an injury or eye surgery are most susceptible. Less frequently, friction between the retina and vitreous or scar tissue pulls the retina loose, something that occurs most often in patients with diabetes. Third, disease-related swelling or bleeding under the retina can push it away from the eye wall.
Signs of retinal tear or detachment include flashes of light, a group or web of floaters, wavy or watery vision, a sense that there is a veil or curtain obstructing peripheral vision, or a sudden drop in vision quality. If you experience any of these symptoms, call your doctor immediately. Early treatment is essential to preserve your vision and is usually done through laser and cryoprobe procedures.
Diabetic retinopathy is a complication of diabetes that weakens the blood vessels that supply nourishment to the retina (the light-sensitive lining in the back of the eye where vision is focused). These weak vessels can leak, swell, bleed or develop thin branches, causing a loss of vision. Changes to your vision may not be noticeable at first. But in its advanced stages, the disease can cause blurred or cloudy vision, floaters and blind spots – and, eventually, blindness. This damage is irreversible. Diabetic retinopathy is the most common diabetic eye complication and a leading cause of blindness in American adults. Macular edema, which is leaking fluid that causes blurred vision, often occurs with diabetic retinopathy.
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Diabetic retinopathy can be classified as either proliferative or nonproliferative. Nonproliferative diabetic retinopathy is the most common type and involves weakened blood vessel walls within the retina that leak fluid or blood into the retina through tiny bulges known as microaneurysms. Proliferative diabetic retinopathy is classified as more severe and involves the growth of new blood vessels within the vitreous that may cause the retina to detach from the back of the eye.
Diabetic retinopathy can affect anyone with diabetes, although it is more likely to occur in patients who:
When patients with diabetes do not properly control their blood sugar levels, the natural lens may swell and begin to leak blood or fluid into the retina. This leaking and swelling can cause blurred vision, and with too much sugar in the blood, may be unable to properly nourish the retina.
Many patients do not experience any symptoms while diabetic retinopathy is in its early stages. As the condition progresses, patients may experience:
If left untreated, diabetic retinopathy can cause permanent vision loss and even blindness.
Treatment for diabetic retinopathy depends on the type and severity of the condition, but may include just monitoring the condition to make sure it does not progress too quickly. More severe cases, including proliferative diabetic retinopathy, may require laser treatment or surgery. Laser treatment may be done through photocoagulation to stop the leakage of blood and fluid or panretinal photocoagulation to shrink the abnormal blood vessels. Vitrectomy may also be performed to surgically remove blood from the center of the eye.
Although most flashes and floaters occur in people with healthy or merely nearsighted eyes, they can be symptoms of serious problems including injury and retinal and posterior vitreous detachments. Flashes in vision are caused by traction on the retina, the bundle of nerves in the back of the eye where images are detected and transmitted to the brain. Patients complain of flashing lights or lightning streaks.
Floaters are often seen when fibers move within the vitreous humor, the gelatinous substance made of water and protein fibers that fills the eye. Patients complain of small specks or dots that can be seen against clear backgrounds. Serious vision loss can occur if the retina detaches from the eye wall. Patients experiencing flashes and floaters should contact their doctor immediately so an examination can be performed.
A vitreous hemorrhage involves a rupturing and leaking of the retinal blood vessels into the vitreous humor. This is most often caused by weakened blood vessels in the retina, and may be associated with diabetic retinopathy. Patients with a vitreous hemorrhage may experience dark spots in their vision or completely obstructed vision as the amount of light that can pass through the vitreous is reduced. These symptoms may be present for several months before the body reabsorbs the blood.
Treatment for a vitreous hemorrhage may be limited to “watchful waiting” to see if the hemorrhage will clear on its own, or may include vitrectomy, a surgical procedure to remove the vitreous humor and replace it with a balanced salt solution. Your doctor will decide which option is best for you based on your individual condition.
The macula is the part of the retina in the back of the eye that ensures that our central vision is clear and sharp. As you age, the tissue in the eye responsible for central vision slowly begins to deteriorate, significantly affecting a patient’s quality of life and causing a condition known as macular degeneration. Also known as age-related macular degeneration (AMD), this condition is common among older adults and is the leading cause of vision loss and blindness in people over the age of 65.
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Macular degeneration can be classified as either wet (neovascular or exudative) or dry (non-neovascular or non-exudative). Dry macular degeneration is the more common diagnosis, and is considered to be an early stage of the disease. This type of disease usually develops as a result of aging and thinning of the macular tissues and the clumping or depositing of pigment within the macula.
Only about 10% of patients see their condition progress to the more advanced and damaging wet macular degeneration. In wet macular degeneration, new blood vessels develop beneath the retina and cause a leakage of blood and fluid. This leakage can lead to permanent damages in the central vision and the creation of blind spots. Although less common, wet macular degeneration accounts for 90% of blindness caused by all cases of this condition.
Macular degeneration is most common in females and whites, although the risk for all patients increases with age. This condition is the leading cause of blindness in the US for patients over the age of 65.
Other risk factors for developing macular degeneration include:
Many cases of macular degeneration are a result of aging and the natural deterioration of eye tissue that is needed for clear vision. This disease can also be related to a genetic factor in patients who have a gene variant known as complement factor H. Nearly half of the blinding cases of macular degeneration are linked to this genetic deficiency. Variants of another gene, called complement factor B, may also be involved in the development of macular degeneration.
Patients with macular degeneration may notice gradual changes to their vision, including shadowy areas in the central vision or fuzzy and distorted vision. These areas grow larger as the disease progresses, and can eventually turn into blind spots. Patients may also have difficulty seeing color and fine details.
If the disease progresses to the wet form, patients may also see straight lines as wavy. Central vision loss may occur rapidly, sometimes in as little as a few days or weeks.
While there is no cure for macular degeneration, there are several treatment options available to help patients manage this condition and preserve their vision. The best treatment option for each patient depends on the severity and type of the condition, as well as how much, if any, permanent vision loss has occurred.
Intraocular injections of Avastin, Lucentis and Macugen are often successful in stopping abnormal blood vessel growth in wet macular degeneration. These FDA-approved medications are injected into the vitreous of the eye on a monthly basis to control the damaging effects of wet macular degeneration. Photodynamic therapy is also effective in removing newly developing abnormal blood vessels that are characteristic of wet macular degeneration. Many patients also benefit from vitamin and mineral supplements, which can clear out toxic substances that may build up in advanced cases of this condition.
Patients who are at a higher risk of developing macular degeneration can take certain measures to prevent the disease from developing. While there is no one foolproof method for preventing macular degeneration, patients can:
A retinal vein occlusion occurs when an artery supplying blood to the eye hardens or swells and presses on a nearby vein and blocks it, making it difficult for blood to leave the eye. The restricted circulation leads to high pressure in the eye, which can in turn cause swelling, bleeding, growth of abnormal blood vessels, and partial or total vision loss.
Vascular occlusions do not cause a change in physical appearance, and they can occur with no pain or noticeable loss of vision. For these reasons, it is important to have routine eye exams and also to check one’s own vision by closing one eye at a time.
There is no cure, so emphasis is placed on risk management, treatment of symptoms and prevention of further vision loss. It is critical to control high blood pressure, high cholesterol, diabetes and other health conditions that increase the risk of vascular hardening, narrowing and blood clotting.
Ophthalmologists Dr. John E. Godard,M.D., Dr. Roger Rossomondo,M.D., Dr. G. Keith Berry,M.D.,
Dr. John Thomas,M.D., Dr. Evan S. Loft, M.D., and Dr. Eric Baylin, M.D. serving Carrollton, Villa Rica,
Bremen, Newnan, LaGrange, Douglasville, Anniston AL, Lithia Springs, Rome, Peachtree and the surrounding area.
Carrollton Eye Clinic | 158 Clinic Avenue | Carrollton, GA 30117 | Phone: 770-834-1008 | www.carrolltoneye.com
Villa Rica Eye Clinic | 705 Dallas Highway | Villa Rica, GA 30180 | Phone: 770-459-2022
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